Introduction

Rapid sequence intubation (RSI) is a process whereby an induction agent and a neuromuscular blocking agent are given in rapid succession to facilitate endotracheal intubation

The immediate post intubation period in the ED is a critical time for continued patient stabilization.

Key Points

  • The immediate post-intubation period in the ED is a critical time for continued patient stabilization.
  • Do not let an ongoing paralytic mask the need for sedation — a still patient may still be in pain or distress.
  • An analgesia-first (analgosedation) approach can be built from common agents reviewed here.
  • The cited evidence favors lighter over deeper sedation and non-benzodiazepine strategies.

Clinical Detail

Depending on the paralytic used, clinicians can be eased into the assumption that the patient is tolerating the ventilator and not in need of sedation or analgesia.

Administering analgesia and sedation is key to preventing patient awareness during paralysis and preventing PTSD. The agents below summarize dosing, pharmacokinetics, and safety considerations for common post-intubation analgesia and sedation options.

ParameterFentanyl (Sublimaze)Propofol (Diprivan)Midazolam (Versed)Dexmedetomidine (Precedex)
DoseBolus: 0.35 to 1.5 mcg/kg IV every 0.5 to 1 hour
Infusion: 25–300 mcg/hr
Bolus:
Infusion: Titrate in 5–50 mcg/kg/min
Bolus: 0.5 to 4 mg
Infusion: 1–10 mg/hr
Bolus: Not recommended
Infusion: 0.1–1.4 mcg/kg/hr
AdministrationIV Bolus + InfusionIV Bolus + InfusionIV Bolus + InfusionIV Infusion
PK/PDOnset: IV almost immediate
Duration: 30–60 min
Metabolism: CYP3A4
Excretion: >90% inactive metabolite renally eliminated
Onset: 10–40 sec
Duration: 3–10 min
Metabolism: Hepatic Phase II
Excretion: Urine (~88% metabolites)
Onset: 3–5 min
Duration: 30–80 min
Metabolism: CYP3A4 (active metabolites)
Excretion: 45% to 57% renally eliminated (metabolites)
Onset: 15–30 min
Duration: 4 hours
Metabolism: Hepatic Phase II + CYP2A6
Excretion:
Adverse EffectsChest wall rigidity, CNS depressionHypotension, bradycardia, hypertriglyceridemiaHypotension, respiratory depressionHypotension, bradycardia
Drug InteractionsCYP3A4 inhibitors, serotonergic agentsBupivacaine, St. John’s WortCYP3A4 inhibitors, CNS depressantsCNS depressants and antihypertensive
CompatibilityProtonixCalcium chloride, Nimbex, Cipro, gentamicin, phenytoinFosphenytoin, sodium bicarbonate, Zosyn, hydrocortisoneProtonix, phenytoin

Evidence

Author, YearDesign / Sample SizeIntervention & ComparisonOutcome
Groetzinger, 2018Retrospective review / n=91Ketamine infusion 0.125 to 1.2 mg/kg/hr63% of patients discontinued other sedatives or analgesic within 24 hours of initiating ketamine; ↑ in the number of sedation scores at goal; ↓ in agitation, defined as SAS >4, after the initiation of ketamine
Shehabi, 2018Prospective cohort / n=703Light Sedation vs Deep Sedation (using sedation intensity score)Sedation intensity independently, in an ascending relationship, predicted increased risk of death, delirium, and delayed time to extubation
Fraser, 2013Meta-analysis / n=1,235 patientsBenzodiazepines vs Non-benzodiazepinesNon-benzodiazepine sedatives associated with ↓ ICU LOS and ↓ ventilator days
Amini, 2013Retrospective cohort study / n=100Rocuronium-assisted RSI + PharmD vs Rocuronium-assisted RSI − PharmDPharmD associated with time to sedation 9 min vs 28 min; PharmD associated with time to analgesia 21 min vs 44 min; PharmD associated with sedation within 5 min 33% vs 11%
Watt, 2012Retrospective cohort study / n=200Succinylcholine 1.7 ± 0.7 mg/kg vs Rocuronium 1.3 ± 0.4 mg/kgAfter intubation, 77.5% (n=155) of patients were initiated on a sedative infusion of propofol (n=148) or midazolam (n=7). Mean time to post-intubation sedation was significantly greater with rocuronium compared to succinylcholine (27 min vs 15)
Shehabi (SPICE), 2012Prospective cohort / n=251Light Sedation vs Deep Sedation4 hours after starting mechanical ventilation 76% of patients were deeply sedated (RASS −3 to −5); early deep sedation was a significant independent predictor of death and time to extubation
Jakob, 2012RCT / N=498Dexmedetomidine 0.2–1.4 mcg/kg/hr vs Midazolam 0.03–0.2 mg/kg/hrLighter sedation, fewer ventilation days
Strom, 2010RCT / n=140No sedation (PRN morphine) vs Propofol or midazolam infusion + PRN morphineNo sedation group had ↓ ventilator days, ↓ ICU LOS, and ↓ hospital LOS

Conclusions

  • The immediate post-intubation period is a critical time for continued stabilization, and providing analgesia and sedation is key to preventing patient awareness during paralysis and preventing PTSD.
  • Do not let an ongoing paralytic mask the need for sedation: a still patient may appear to tolerate the ventilator while remaining aware, so analgesia and sedation should not be withheld simply because the paralytic is still working.
  • An analgesia-first (analgosedation) approach can be built from common agents reviewed here, fentanyl, propofol, midazolam, and dexmedetomidine, selected by their dosing, pharmacokinetics, adverse effects, and compatibility.
  • The cited evidence favors lighter over deeper sedation and non-benzodiazepine strategies, and pharmacist involvement at the bedside was associated with faster time to post-intubation sedation and analgesia.

References

Micromedex [Electronic version].Greenwood Village, CO: Truven Health Analytics. Retrieved September 6, 2018, from http://www.micromedexsolutions.com/

Groetzinger LM. Pharmacotherapy. 2018 Feb;38(2):181-188

Jakob SM. JAMA. 2012 Mar 21;307(11):1151-60.

Shehabi Y. (SPICE Investigators). Am J Respir Crit Care Med. 2012 Oct 15;186(8):724-31

Watt JM. Emerg Med J. 2013 Nov;30(11):893-5.

Amini A. Am J Health Syst Pharm. 2013 Sep 1;70(17):1513-7.

Fraser GL. Crit Care Med. 2013 Sep;41(9 Suppl 1):S30-8.

Shehabi Y. Crit Care Med. 2018 Jun;46(6):850-859.

Tags: post-intubation analgesia sedation RSI

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