Introduction

  • The overall prevalence of ACE inhibitor angioedema is low (~0.2-0.7%), however it makes up the
  • majority of visits to the ED for angioedema

  • ACE inhibitor angioedema is caused by the buildup of bradykinin which leads to increased vascular
  • permeability and the release of substance P resulting in vasodilation and fluid extravasation into

    tissues

  • ACE inhibitor angioedema can occur at any time, with the majority of cases occurring within the
  • first month of therapy.

  • Risk factors for ACE inhibitor angioedema includes female sex, African American race, chronic
  • heart failure, coronary artery disease, history of smoking, and concomitant use of dipeptidyl

    peptidase 4 (DPP4) inhibitors

Clinical Detail

Fresh Frozen Plasma (FFP)IcatibantTranexamic Acid (TXA)
Dose2-4 units30 mg1000 mg
AdministrationIntravenousSubcutaneousIntravenous
PK/PDOnset: ~ 2 hoursOnset: 2 hours
Duration: 6 hours
Onset: ~ 2 hours
Adverse EffectsHypervolemia, TRALI, thrombosis, hyperfibrinolysis, infectionInjection site reactions, LFT elevations, dizzinessAbdominal pain, headache, musculoskeletal pain/spasms
Drug Interactions and warningsCalcium administration within the same line (may produce precipitants)NoneContraindicated in patients with SAH or active intravascular clotting
CompatibilityN/AN/ACompatible with NS, D5W, or LR
CommentsTakes ~ 30-45 minutes to thaw & process once orderedCosts ~ $4,500 per 30 mg syringe (AWP)Available in most emergency departments. Inject 1000 mg TXA in 100 mL of diluent

Evidence

Author, yearDesign / sample sizeIntervention & ComparisonOutcome
Baş, 2015RCT (n = 27)SubQ Icatibant 30 mg vs. prednisolone + clemastineTime to complete symptom resolution: 8 hours vs. 27.1 hours (P=0.002).

Time to onset of symptom relief: 2 hours vs. 11.7 hours (P=0.03).

All patients experienced complete resolution of edema.
Straka, 2017RCT (n = 33)SubQ Icatibant 30 mg vs placeboTime-to-resolution (27.2 hrs vs 35.3 hrs) and amount of swelling over time were similar in placebo and icatibant treatment groups.
Sinert, 2017RCT (n = 121)SubQ Icatibant 30 mg vs placeboThere were no difference time to meeting discharge criteria between groups (4 hrs vs 4 hrs).
Karim, 2002Case Report (n = 1)4 units FFP following chlorpheniramine, hydrocortisone, and epinephrineDramatic improvement within 2 hours following FFP administration allowing for extubation.
Warrier, 2004Case Report (n = 1)2 units FFP following antihistamine, corticosteroid, epinephrine, antileukotrienes, cyclosporine, and IVIG (all without improvement)Complete resolution of symptoms within 2-4 hours following FFP administration.
Bolton, 2012Case Report (n = 1)2 units FFP following antihistamine and corticosteroid administrationNear complete resolution of symptoms within 2 hours following FFP administration.
Hassen, 2013Case series (n = 7)2 units FFP following antihistamine and corticosteroid administrationTemporal association between the administration of FFP and improvement in angioedema in 7 cases of presumed ACEI-induced angioedema that were refractory to histamine-related anaphlaxis.
Stewart, 2013Case Report (n = 2)2 units FFP administeredAdministration of FFP resulted in rapid resolution of symptoms in both patients.
Beauchêne, 2018Case Series (n = 33)Tranexamic Acid
IV: 24 patients (73%)
PO: 8 patients (24%)
Unknown: 1 patient (3%)

Dosage: 500 mg – 4 grams (55% received 1 g)
81.8% patients achieved significant improvement following TXA administration alone.

39.3% patients experienced symptom improvement within 1 hr of TXA administration.
Wang, 2020Case Report (n = 1)TXA 1000 mg IVPB over 10 minutes administered following diphenhydramine, famotidine, methylprednisolone, and epinephrineImprovement in speech observed within 30 minutes of TXA administration with complete resolution of symptoms at 2 hrs.

Patient discharged 2.5 hours following presentation to ED.
Manzano, 2021Case series (n = 11)TXA 1000 mg IVThe median length of stay in the hospital was 1.2 days (0.4-18.2 days).

No noted adverse effects related to medication administration of TXA.

Conclusions

    (Christian Leppert & [email protected]

  • Many cases of ACE inhibitor angioedema will resolve on their own with the cessation of the
  • offending agent.

  • More studies are needed to evaluate the use of investigational therapies including tranexamic acid
  • and FFP.

  • In severe cases of ACE inhibitor angioedema, it is reasonable to consider using tranexamic acid or
  • FFP if icatibant is unavailable after weighing the risks versus benefits.

References

  • Lexicomp Online, Hudson, Ohio: UpToDate, Inc.; 2021; April 22, 2021. Available from:
  • http://online.lexi.com/lco/action/home

  • Brown T, Gonzalez J, Monteleone C. Angiotensin-converting enzyme inhibitor-induced angioedema: a review
  • of the literature. J Clin Hypertens. 2017;19:1377-1382.

  • Campo P, Fernandez TD, Canto G, et al. Angioedema induced by angiotensin-converting enzyme inhibitors.
  • Curr Opin Allergy Clin Immunol. 2013;13:337-344.

  • Byrd JB, Touzin K, Sile S, et al. Dipeptidyl peptidase IV in angiotensin-converting enzyme inhibitor-associated
  • angioedema. Hypertension. 2008;51:141-147.

  • Straka BT, Ramirez CE, Byrd JB, Stone E, Woodard-Grice A, Nian H, Yu C, Banerji A, Brown NJ. Effect of
  • bradykinin receptor antagonism on ACE inhibitor-associated angioedema. J Allergy Clin Immunol. 2017

    Jul;140(1):242-248.e2. doi: 10.1016/j.jaci.2016.09.051. Epub 2016 Nov 29. PMID: 27913306; PMCID: PMC5705179.

  • Sinert R, Levy P, Bernstein JA, Body R, Sivilotti MLA, Moellman J, Schranz J, Baptista J, Kimura A, Nothaft W;
  • CAMEO study group. Randomized Trial of Icatibant for Angiotensin-Converting Enzyme Inhibitor-Induced

    Upper Airway Angioedema. J Allergy Clin Immunol Pract. 2017 Sep-Oct;5(5):1402-1409.e3. doi:

  • 1016/j.jaip.2017.03.003. Epub 2017 May 25. PMID: 28552382.
  • Baş M, Greve J, Stelter K, et al. A randomized trial of icatibant in ACE-inhibitor-induced angioedema. N Engl J
  • Med. 2015;372:418-425.

  • Karim MY, Masood A. Fresh-frozen plasma as a treatment for life-threatening ACE-inhibitor angioedema. J
  • Allergy Clin Immunol. 2002;109(2):370-1.

  • Warrier MR, Copilevitz CA, Dykewicz MS, et al. Fresh frozen plasma in the treatment of resistant angiotensin-
  • converting enzyme inhibitor angioedema. Ann Allergy Asthma Immunol. 2004;92:573-575

  • Hassen GW, Kalantari H, Parraga M, Chirurgi R, Meletiche C, Chan C, Ciarlo J, Gazi F, Lobaito C, Tadayon S,
  • Yemane S, Velez C. Fresh frozen plasma for progressive and refractory angiotensin-converting enzyme

    inhibitor-induced angioedema. J Emerg Med. 2013 Apr;44(4):764-72. doi: 10.1016/j.jemermed.2012.07.055.

    Epub 2012 Oct 28. PMID: 23114109.

  • Bolton MR, Dooley-Hash SL. Angiotensin-converting enzyme inhibitor angioedema. J Emerg Med.
  • 2012;43(4):e261-262.

  • Stewart M, McGlone R. Fresh frozen plasma in the treatment of ACE inhibitor-induced angioedema. BMJ Case
  • Rep. 2012;2012:bcr2012006849.

  • Wang K, Geiger H, McMahon A. Tranexamic acid for ACE inhibitor induced angioedema – A case report. Am
  • J Emerg Med. 2020;S0735-6757(20)30923-2.

  • Beauchêne C, Martins-Héricher J, Denis D, et al. Tranexamic acid as first-line emergency treatment for
  • episodes of bradykinin-mediated angioedema induced by ACE inhibitors. Rev Med Interne. 2018;39(10):772-

  • Martinez Manzano JM, Lo KB, Patarroyo-Aponte G, Azmaiparashvili Z. The use of intravenous tranexamic acid
  • for patients with angiotensin-converting enzyme inhibitor-induced angioedema: A case series. Ann Allergy

    Asthma Immunol. 2021 Feb 14:S1081-1206(21)00129-0. doi: 10.1016/j.anai.2021.02.011. Epub ahead of print.

    PMID: 33592284.

Tags:ACE inhibitor angioedema bradykinin icatibant